Chapter 1 Chairman's creation (pages 1–3): L. Wolpert
Chapter 2 regulate of Early Embryonic center Morphogenesis: A speculation (pages 4–19): Francis J. Manasek
Chapter three styles of Vascularization within the constructing Cerebral Cortex (pages 20–36): Thomas Bar
Chapter four The development of Elastin within the Aorta and massive Arteries of Mammals (pages 37–55): Margot R. Roach
Chapter five ‘Structural Autoregulation’ — The neighborhood variation of Vascular Beds to power alterations in strain (pages 56–79): E. R. Pfefferkorn and Bjorn Folkow
Chapter 6 actual elements and Angiogenesis (pages 80–99): Terence J. Ryan and Raymond L. Barnhill
Chapter 7 Vascularization of Experimental Tumours (pages 100–119): H.S. Reinhold and A. Van Den Berg?Blok
Chapter eight function of Mast Cells in Experimental Tumour Angiogeneslis (pages 120–131): R. Ashley Fraser and John G. Simpson
Chapter nine The position of Heparin in Angiogenesis (pages 132–149): Judah Folkman, Stephanie Taylor and Curt Spillberg
Chapter 10 Degradation of Basement Membrane parts through Vascular Endothelial Cells: position in Neovascularization (pages 150–162): Bertm Glaser, Tia Kalebic, S. Garbisa, Thomas B. Connor and Lance A. Liotta
Chapter eleven Inhibition of Neovascularization through a Cartilage issue (pages 163–206): Klaus E. Kuettner and Bendicht U. Pauli
Chapter 12 Pathological Vascularization of the Coronary Intima (pages 207–221): W. J. Cliff and G. I. Schoefl
Chapter thirteen Maldevelopments of the Vascular procedure: scientific Conundrums (pages 222–243): Anthony E. younger
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Additional info for Ciba Foundation Symposium 100 - Development of the Vascular System
Thus it is impossible to say whether the modulus of the elastin itself changes or whether the alterations are due to the addition of more layers of elastin or to changes in the fenestrations or other components of the wall. The development of branches Obviously the cross-sectional area of the vascular tree increases enormously between the aorta and the capillaries, but the exact way this change occurs at each branch is still unknown. Zamir (1976) has used optimality principles to determine the most advantageous angles and diameters in peripheral branches and has shown experimentally that his predictions fit the observed patterns in a number of vascular beds (Zamir & Brown 1982).
This agrees with the marked decrease in the incorporation of [3H]thymidine which has been observed to occur between postnatal days 10 and 15 (see Mare5 & Briickner 1978, Ichikawa & Hirata 1982). e. between days 14 and 20). This coincides in time with the decrease in the pericytic circumferential coverage of the capillaries. That is, the area of direct contact between E C and PC is reduced. After the second postnatal week no mitotic division of PC in situ is observed. The total number of PC remains fairly constant.
The counts in the two different preparations yielded similar results. ) The number of groups may vary according to the final cortical thickness (Fig. 1)* The branching or sprouting process becomes more restricted to the distal (capillary) segments of the vascular tree as the cortex matures. The numerical density of vascular branching increases to maximum values during the first three weeks of postnatal life (Table 2). 05) than those present at three weeks after birth. This reduction is inversely related to the increase in tissue volume during late postnatal growth.
Ciba Foundation Symposium 100 - Development of the Vascular System